On T1-weighted images without fat suppression, the pancreas is isointense or slightly hyperintense to the liver. With fat suppression, the normal pancreas is hyperintense to the liver or other solid organs in the abdomen. High signal intensity on T1-weighted images is due to the aqueous protein in the glandular tissue . 2 On T2-weighted images, the pancreas has similar signal intensity to the liver, and is hypointense compared with the spleen or kidneys. After administration of contrast material, the pancreas enhances maximally during the pancreatic phase in contrast to the liver, which shows minimal enhancement during this phase because of its predominant portal venous supply. During the portal and delayed phases, the pancreas becomes isointense to the liver
Normal MR appearance of the pancreas
On T1-weighted images without fat suppression, the pancreas is isointense or slightly hyperintense to the liver. With fat suppression, the normal pancreas is hyperintense to the liver or other solid organs in the abdomen. High signal intensity on T1-weighted images is due to the aqueous protein in the glandular tissue . 2 On T2-weighted images, the pancreas has similar signal intensity to the liver, and is hypointense compared with the spleen or kidneys. After administration of contrast material, the pancreas enhances maximally during the pancreatic phase in contrast to the liver, which shows minimal enhancement during this phase because of its predominant portal venous supply. During the portal and delayed phases, the pancreas becomes isointense to the liver
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